OxLDL plays a major role in the initiation and progression of atherosclerotic lesions even though further factors are needed to promote fibrous cap rupture and thrombotic occlusion of the arterial lumen. Pathogens have been implicated in this process but it remains unclear how they can cooperate with oxLDL in amplifying the destructive inflammatory response.
Monteleone, I., Muscoli, S., Terribili, N., Zorzi, F., Mariano, E., Mehta, J., et al. (2013). Local immune activity in acute coronary syndrome: oxLDL abrogates LPS-tolerance in mononuclear cells isolated from culprit lesion. INTERNATIONAL JOURNAL OF CARDIOLOGY, 169(1), 44-51 [10.1016/j.ijcard.2013.08.082].
Local immune activity in acute coronary syndrome: oxLDL abrogates LPS-tolerance in mononuclear cells isolated from culprit lesion
MONTELEONE, IVAN;MUSCOLI, SAVERIO;TERRIBILI, NOEMI;PALLONE, FRANCESCO;MONTELEONE, GIOVANNI;ROMEO, FRANCESCO
2013-10-25
Abstract
OxLDL plays a major role in the initiation and progression of atherosclerotic lesions even though further factors are needed to promote fibrous cap rupture and thrombotic occlusion of the arterial lumen. Pathogens have been implicated in this process but it remains unclear how they can cooperate with oxLDL in amplifying the destructive inflammatory response.File in questo prodotto:
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