To investigate the effects of TLR4 antagonism on human endothelial cells activation and cytokine expression, and whether the Asp299Gly TLR4 polymorphism is associated with better endothelial function in patients with rheumatoid arthritis (RA).
Menghini, R., Campia, U., Tesauro, M., Marino, A., Rovella, V., Rodia, G., et al. (2014). Toll-like receptor 4 mediates endothelial cell activation through NF-κB but is not associated with endothelial dysfunction in patients with rheumatoid arthritis. PLOS ONE, 9(6), e99053-e99053 [10.1371/journal.pone.0099053].
Toll-like receptor 4 mediates endothelial cell activation through NF-κB but is not associated with endothelial dysfunction in patients with rheumatoid arthritis
MENGHINI, ROSSELLA;TESAURO, MANFREDI;ROVELLA, VALENTINA;DI DANIELE, NICOLA;FEDERICI, MASSIMO;
2014-01-01
Abstract
To investigate the effects of TLR4 antagonism on human endothelial cells activation and cytokine expression, and whether the Asp299Gly TLR4 polymorphism is associated with better endothelial function in patients with rheumatoid arthritis (RA).File in questo prodotto:
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