The aim of this study was to determinate the possible role of the ionotropic glutamate receptor in the expression of irreversible electrophysiological changes induced by in vitro ischemia and to test whether the neuroprotective action of various neurotransmitter agonists and drugs of clinical interest is related to a presynaptic inhibitory action at glutamatergic synapses.
Calabresi, P., Picconi, B., Saulle, E., Centonze, D., Hainsworth, A., Bernardi, G. (2000). Is pharmacological neuroprotection dependent on reduced glutamate release?. STROKE, 31(3), 766-772 [10.1161/01.STR.31.3.766].
Is pharmacological neuroprotection dependent on reduced glutamate release?
CALABRESI, PAOLO;CENTONZE, DIEGO;BERNARDI, GIORGIO
2000-03-01
Abstract
The aim of this study was to determinate the possible role of the ionotropic glutamate receptor in the expression of irreversible electrophysiological changes induced by in vitro ischemia and to test whether the neuroprotective action of various neurotransmitter agonists and drugs of clinical interest is related to a presynaptic inhibitory action at glutamatergic synapses.File in questo prodotto:
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