Striatal spiny neurons are selectively vulnerable to ischemia, but the ionic mechanisms underlying this selective vulnerability are unclear. Although a possible involvement of sodium and calcium ions has been postulated in the ischemia-induced damage of rat striatal neurons, the ischemia-induced ionic changes have never been analyzed in this neuronal subtype.

Calabresi, P., Marfia, G.a., Centonze, D., Pisani, A., Bernardi, G. (1999). Sodium influx plays a major role in the membrane depolarization induced by oxygen and glucose deprivation in rat striatal spiny neurons. STROKE, 30(1), 171-179 [10.1161/01.STR.30.1.171].

Sodium influx plays a major role in the membrane depolarization induced by oxygen and glucose deprivation in rat striatal spiny neurons

CALABRESI, PAOLO;MARFIA, GIROLAMA ALESSANDRA;CENTONZE, DIEGO;PISANI, ANTONIO;BERNARDI, GIORGIO
1999-01-01

Abstract

Striatal spiny neurons are selectively vulnerable to ischemia, but the ionic mechanisms underlying this selective vulnerability are unclear. Although a possible involvement of sodium and calcium ions has been postulated in the ischemia-induced damage of rat striatal neurons, the ischemia-induced ionic changes have never been analyzed in this neuronal subtype.
gen-1999
Pubblicato
Rilevanza internazionale
Articolo
Sì, ma tipo non specificato
Settore MED/26 - NEUROLOGIA
English
Con Impact Factor ISI
Glyburide; Calcium Channels; Rats, Wistar; Sodium Channels; Cerebral Cortex; Organ Culture Techniques; Hypoglycemic Agents; Magnesium; Potassium Channels; Glucose; Corpus Striatum; Calcium Channel Blockers; Sodium Channel Blockers; Potassium Channel Blockers; Egtazic Acid; Chelating Agents; Adenosine Triphosphate; Rats; Brain Ischemia; Animals; Calcium; Sodium; Patch-Clamp Techniques; Receptors, Glutamate; Membrane Potentials; Tetrodotoxin; Oxygen; Neurons; Tolbutamide
Calabresi, P., Marfia, G.a., Centonze, D., Pisani, A., Bernardi, G. (1999). Sodium influx plays a major role in the membrane depolarization induced by oxygen and glucose deprivation in rat striatal spiny neurons. STROKE, 30(1), 171-179 [10.1161/01.STR.30.1.171].
Calabresi, P; Marfia, Ga; Centonze, D; Pisani, A; Bernardi, G
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/2108/51479
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