Several observations indicate that, during energy deprivation, endogenous dopamine may become neurotoxic. Accordingly, the nucleus striatum is a preferential site of silent infarcts in humans, and experimental ischemia caused by homolateral carotid occlusion selectively damages this dopamine-enriched brain area. In an attempt to clarify how dopamine takes part in ischemia-induced neuronal damage, we performed in vitro electrophysiological recordings from neurons of the nucleus striatum.

Saulle, E., Centonze, D., Martín, A., Moratalla, R., Bernardi, G., Calabresi, P. (2002). Endogenous dopamine amplifies ischemic long-term potentiation via D1 receptors. STROKE, 33(12), 2978-2984 [10.1161/01.STR.0000038093.42512.0F].

Endogenous dopamine amplifies ischemic long-term potentiation via D1 receptors

CENTONZE, DIEGO;BERNARDI, GIORGIO;CALABRESI, PAOLO
2002-12-01

Abstract

Several observations indicate that, during energy deprivation, endogenous dopamine may become neurotoxic. Accordingly, the nucleus striatum is a preferential site of silent infarcts in humans, and experimental ischemia caused by homolateral carotid occlusion selectively damages this dopamine-enriched brain area. In an attempt to clarify how dopamine takes part in ischemia-induced neuronal damage, we performed in vitro electrophysiological recordings from neurons of the nucleus striatum.
dic-2002
Pubblicato
Rilevanza internazionale
Articolo
Sì, ma tipo non specificato
Settore MED/26 - NEUROLOGIA
English
Con Impact Factor ISI
Rats, Wistar; Mice, Knockout; Excitatory Postsynaptic Potentials; Excitatory Amino Acid Antagonists; Mice; Perfusion; Dopamine; Denervation; Dopamine Antagonists; Glucose; Corpus Striatum; Cyclic AMP-Dependent Protein Kinases; Benzazepines; Rats; Brain Ischemia; Animals; Receptors, Dopamine D1; Receptors, N-Methyl-D-Aspartate; Oxidopamine; Membrane Potentials; Cyclic AMP; Neurons; Signal Transduction; Long-Term Potentiation; Synaptic Transmission; Energy Metabolism
Saulle, E., Centonze, D., Martín, A., Moratalla, R., Bernardi, G., Calabresi, P. (2002). Endogenous dopamine amplifies ischemic long-term potentiation via D1 receptors. STROKE, 33(12), 2978-2984 [10.1161/01.STR.0000038093.42512.0F].
Saulle, E; Centonze, D; Martín, A; Moratalla, R; Bernardi, G; Calabresi, P
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/2108/50351
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